Herbs are NOT created equally

Herbs are NOT created equally.

Originally posted on Feel good, live healthy:


Benefits of avocados

– To lose weigh
– For clear skin
– Helps burn fat
– To boost immune system
– To increase energy
– To reduce signs of aging
– High vitamin A & C
– Good for diabetes
– To promote heart health
– To lower cholesterol
– Good for arthritis
– They are delicious!

View original

Herbology 101: Ginger

*Ginger has long been employed by folk medicine to treat diarrhea, flatulence, indigestion, and loss of appetite. In Ayurvedic medicine, ginger is called vishwabhesaj, “The Universal Medicine.” Ginger contains a volatile oil and other compounds which act as digestive stimulants to encourage the production of digestive fluids and saliva. Thus, ginger helps reduce the symptoms of improper chewing, overeating, or excessive motion by enabling digestion to be more effective. Ginger also neutralizes acids and toxins in the digestive tract which reduces gas and pain in the bowel.

Ginger has been found helpful for expelling phlegm and is used to treat asthma, bronchitis, colds, coughs and respiratory congestion. Ginger also induces perspiration, making it useful in the treatment of fevers, including malaria.

Ginger has been shown to reduce inflammation by acting as a prostaglandin inhibitor, in much the same way as non-steroidal anti-inflammatory drugs (NSAIDs) work. Studies show ginger relaxes muscle spasms and relieves pain and inflammation associated with rheumatic conditions.

A study published in Lancet showed ginger was an effective natural remedy for motion sickness. Two capsules of ginger taken 20-25 minutes before airflight or before embarking on a ship, followed by two capsules every 4 hours, was proven to work better than 100mg of dimenhydrinate (dramamine). Ginger has also been clinically proven to substantially reduce diarrhea, nausea and vomiting associated with the common 24-hour and three-day flus. Clinical trials conducted at St. Bartholomew’s Hospital in London in 1990, verified ginger’s effectiveness in treating postoperative nausea. In fact, ginger proved to work better than conventional medications. Of course, ginger is also a well-known remedy for morning sickness and may provide relief from nausea following chemotherapy treatments.

Studies have found ginger produces a strong stimulating effect on muscular contractions of the heart, validating ginger’s use for improving overall circulation. Ginger also helps lower serum cholesterol levels, again assisting circulatory improvement. Researchers have determined that ginger significantly reduces platelet aggregation—the tendency of blood cells to stick together or clot—which may help in the prevention of heart attacks.

Scientists have also confirmed ginger’s antimicrobial properties which are known to fight both bacteria and parasites, including flukes, roundworms and tapeworms. A trial study in China found 70% of patients with bacillary dysentery made a full recovery when given ginger. Ginger is also known to fight intestinal infections, including certain types of food poisoning.

According to Chinese medicine, ginger focuses its warming properties downward, toward the lower extremities, including the colon, kidneys, legs, ovaries, prostate and vagina. Ginger is often recommended to women with amenorrhea, menorrhagia and menstrual cramps.

Ginger is regarded by some herbalists as an aphrodisiac and tonic.

Many of ginger’s therapeutic properties are due to the presence of a volatile oil and its oleoresin content. In fact, gingerol, a type of oleoresin, is largely responsible for ginger’s hot or spicy taste, as well as the herb’s stimulating effects. Ginger contains vitamins B3(niacin), B5 (pantothenic acid) and B9 (folic acid), as well as high amounts of the minerals magnesium, manganese, potassium and silicon.

Used topically, ginger cools the heat of inflamed, painful and stiff joints. Ginger increases circulation and reduces arthritis and rheumatism by means of detoxifying the blood. Ginger has even been used to treat headaches and toothaches. Two to three tablespoons of ginger added to hot bath water helps to relax muscles and ease body aches and pain. Putting the ginger in a large tea bag keeps the water free from floating particles of ginger. The Chinese also employ an oil extract of ginger in massage therapy for helping dandruff and earaches.

Dried ginger is more heating than fresh ginger; therefore, Chinese herbalists do not recommend it for those with “excessive heat” such as inflammatory skin conditions, peptic ulcers or other gastrointestinal inflammation, such as is associated with colitis, Crohn’s disease, diverticulitis and irritable bowel syndrome.


I personally use NSP’s ginger to relieve muscle spasms associated with sciatica – selafay

  • Amenorrhea
  • Asthma
  • Muscle spasms
  • Food Poisoning
  • Menorrhagia
  • Nausea – Vomiting
*Reprint from Hart NSP Master Reference
Herb Allure, Inc.

Herbology 101: Milk Thistle


*Milk Thistle 

Milk thistle (Silybum marianum) is used throughout the world and is one of the most commonly prescribed medicinal herbs. Milk thistle’s reputation for protecting the liver has been studied and confirmed by laboratory and clinical research for the last 30 years, resulting in over 200 clinical studies.1,2



The liver is the largest organ of the body, with numerous essential functions to perform. The liver filters blood from the intestines, transforms toxic compounds into non-toxic substances, produces bile, inactivates pathogenic (disease-causing) microorganisms, synthesizes essential nutrients and compounds, regulates glucose levels, activates/deactivates hormones, stores fat-soluble vitamins, and provides a significant amount of the body’s heat and energy. Unfortunately, damage to the liver can adversely affect many of these important functions, causing a profound deterioration of one’s health—poor liver health directly affects metabolic diseases such as arthritis, diabetes, obesity and thyroid dysfunction. Incidentally, the most common cause of liver damage is fat accumulation in the liver, known as fatty liver. Left untreated, fatty liver can progress to liver inflammation, fibrosis, hepatitis and cirrhosis. The American Liver Foundation estimates that over 50% of the population age 50+ has a “fatty liver.”2-4

Milk thistle demonstrates significant hepatoprotective (liver-protective) activity by stabilizing cellular membrane permeability and directly preventing toxic damage to hepatic cells; by providing antioxidant activity, including increasing intracellular concentration of glutathione; by facilitating cellular regeneration of damaged hepatocytes; and by inhibiting the synthesis of key chemical mediators of inflammation (i.e. leukotrienes and prostaglandins). Milk thistle has also been shown to facilitate production of hydrochloric acid, pancreatic enzymes and bile; improve the flow of bile through the biliary tract; stimulate peristalsis; and improve immune function. Furthermore, recent animal research on milk thistle has identified anti-inflammatory, anticarcinogenic and hypocholesterolemic (cholesterol-lowering) effects.3-14

Most of the pharmacological research conducted on milk thistle has focused on a complex of flavonolignans collectively known as silymarin, which has been identified as the active constituent. Silymarin is one of the most potent hepatoprotective (liver-protecting) substances known. Its anti-hepatotoxic activity has been proven against a variety of liver toxins, including the severe poisoning of Amanita phalloides (the deathcap or toadstool mushroom), the quickest-acting and most virulent of liver toxins.3-6,10

Silymarin not only protects the liver against toxic damage, but it also regenerates hepatocytes (parenchymal cells of the liver) by increasing the rate of RNA synthesis, which stimulates protein synthesis and accelerates cell-regeneration and hepatocyte formation. In other words, silymarin actually increases the production of new liver cells to replace damaged cells. Fortunately, silymarin has not been shown to have stimulatory effect on malignant liver tissue.1-3,5-10

Recognized as a powerful antioxidant, silymarin increases intracellular antioxidant activity, protecting liver cells from free-radical damage. Silymarin’s antioxidant activity is many times more potent than vitamin E. Silymarin has been shown to significantly enhance activity of the antioxidant enzyme SOD (superoxide dismutase) and prevent the depletion of glutathione—a major intrinsic antioxidant used to detoxify drugs such as acetominophen, hormones and chemicals. Tissue depletion of glutathione is an important factor in cell damage and is typically induced by moderate-to-heavy alcohol consumption. By facilitating glutathione synthesis, silymarin increases glutathione levels in digestive tract and hepatic tissues, thereby preventing oxidant-induced cell damage and providing the liver with a greater capacity for detoxification. Furthermore, silymarin has even been shown to increase glutathione levels in healthy subject by more than 35%.1-3,5,10,13,15-17

The German Commission E also approves milk thistle extract (standardized to 70-80% silymarin) for toxic liver damage and as a supportive treatment in chronic inflammatory liver disease and hepatic cirrhosis.5,8,9

Additional clinical indications for milk thistle use, supported by trials using a standardized milk thistle extract (containing 70-80% silymarin), include abnormal liver function, acute and chronic viral hepatitis, alcoholic and non-alcoholic cirrhosis, cholangitis (inflammation of bile ducts) and pericholangitis (inflammation surrounding bile ducts), cholelithiasis (gallstones), cholestasis (impaired bile flow) and subclinical cholestasis of pregnancy, diabetes secondary to cirrhosis, fatty deposits in the liver, liver damage caused by toxic chemical exposure (anaesthesia, drugs, glues, halogenated hydrocarbons, paints, solvents), and deathcap mushroom poisoning. Milk thistle may also help psoriasis by reducing levels of circulating endotoxins and inhibiting leukotriene formation. Furthermore, milk thistle is especially well-indicated in patients with cancer, who are also undergoing chemotherapy, and in HIV-infected individuals on multi-drug protocol.2,3,6,10,13,15,18

A 4-week, double-blind, controlled study was conducted to determine the effects of milk thistle extract on 97 patients with elevated liver enzyme levels, representing slight acute and subacute liver disease, mostly induced by alcohol abuse. Patients receiving 420mg of milk thistle extract daily demonstrated a statistically significant greater decrease in liver enzymes than did the control group. In addition, normalization of hepatocellular changes occurred significantly more often in patients receiving milk thistle extract.3,9,10,19

A recent 12-month study found milk thistle extract to be effective for diabetes complicated by cirrhosis. Diabetic patients with cirrhosis often require insulin treatment due to insulin resistance—a cirrhotic liver takes up less glucose, causing high insulin levels in the blood, which can lead to insulin resistance. After 4 months of treatment (200mg of silymarin, 3 times daily, two hours after each meal), patients receiving silymarin demonstrated a significant decrease in fasting blood glucose levels, mean daily glucosuria levels (urine sugar levels), fasting insulin levels, and mean exogenous insulin requirements (patients’ daily need for insulin decreased). In addition, total cholesterol levels were reduced significantly in the silymarin group. There were no such changes in the untreated group.20,21

Furthermore, a double-blind, randomized, controlled clinical trial found that silymarin significantly decreased patient mortality from liver cirrhosis, especially for patients with alcohol-induced cirrhosis. Silymarin is believed to reduce the metabolic or toxic effects of alcohol on the liver, as well as reduce hepatocellular necrosis (cell/tissue death) which, in turn, may postpone or prevent hepatic failure. Results of the 41-month trial involving 170 patients found that the survival rate among the silymarin-treated group was 49% higher than the placebo group—the reduction in mortality was most pronounced in the alcoholic cirrhosis subgroup. This study also confirmed long-term treatment with milk thistle to be beneficial, particularly in cases of alcohol-induced liver damage.3,9,10,22

An important consideration for milk thistle use is its ability to protect individuals from medication-induced liver damage. In one study, milk thistle was shown to provide significant liver protection (as measured by serum liver enzyme levels) in psychiatic patients who were also taking phenothiazines or butyrophenones. Milk thistle demonstrated no interference with the efficacy of the antidepressants. Another study found that milk thistle reduced the gastrointestinal disorders and side effects experienced by Alzheimer’s patients taking the drug tacrine. Thus, concomitant use of milk thistle may improve drug tolerance and efficacy and serve to prevent liver damage during long-term drug therapy.3,9,10,13,17,23

Milk thistle extract has demonstrated no signs of toxicity and thus, may be used until clinical improvement is noted. There is no restriction on long-term use and no known contraindications or interactions. Milk thistle has also been determined safe for use during pregnancy and lactation. In fact, research suggests that milk thistle use may prevent and correct liver damage during pregnancy. However, due to the herb’s choleretic activity and depending on dosage, milk thistle may produce a mild laxative effect as a result of increased bile flow and secretion.2,3,6,8,10,14,24

NSP’s Time-Release Milk Thistle provides 350mg of milk thistle seed extract per tablet, standardized to 80% silymarin (providing 280mg silymarin). Since research indicates that silymarin absorption is enhanced by lecithin, simultaneous use of lecithin is recommended.6



  • Alcoholism – Fatty Liver
  • Blood Purifiers
  • Cholesterol (reduce)
  • Cirrhosis
  • Detoxification
  • Gallstones
  • Hepatitis
  • Jaundice
  • Liver – Damage
  • Liver – Enlarged
  • Liver – Fatty Liver
  • Liver Health
  • Psoriasis
  • Radiation – Chemotherapy
  • Toxicity – Environmental Pollution



1Alschuler ND, L. “Digestive Disturbances: The Fatty Liver Connection.” International Journal of Integrative Medicine; 2000, 2(2): 16-20.

2Wassef RPh, F. “Enhancing liver detoxification.” American Journal of Natural Medicine; 1998, 5(9):24-27.

3Alschuler ND, L. “Milk Thistle: Goals & Objectives.” International Journal of Integrative Medicine; 1999, 1(1): 29-34.

4Buhner, S. H. Herbs for Hepatitis C and the Liver.Pownal, VT: Storey Books, 2000.

5Hobbs LAc, C. “Milk thistle therapy.” Herbs For Health; 1997, 2(3): 47-49.

6Mills, S. & Bone, K. Principles and Practice of Phytotherapy. London: Churchill Livingstone, 2000.

7Flora, K., et. al. “Milk thistle (Silybum marianum) for the therapy of liver disease.” American Journal of Gastroenterology; 1998, 93(2): 139-143.

8The Complete German Commission E Monographs. Austin, TX: American Botanical Council, 1999.

9Herbal Medicine: Expanded Commission E Monographs. Newton, MA: Integrative Medicine Communications, 2000.

10Pizzorno ND, J. & Murray ND, M. Textbook of Natural Medicine, 2nd ed. London, England: Churchill Livingstone, 1999.

11Manna, S.K., et. al. “Silymarin suppresses TNF-induced activation of NF-kappa B, c-Jun N-terminal kinase, and apoptosis.” Journal of Immunology; 1999, 163(12): 6800-6809.

12Skottova, N. & Krecman, V. “Silymarin as a potential hypocholesterolaemic drug.” Physiological Research; 1998, 47(1): 1-7.

13Miller PhD, L. & Murray PhD, W. Herbal Medicinals. Binghamton, NY: Pharmaceutical Products Press, 1998.

14Deak, G., et. al. [Immunomodulator effect of silymarin therapy in chronic alcoholic liver diseases]. Orvosi Hetilap; 1990, 131(24): 1291-1296.

15Murray ND, M. The Healing Power of Herbs. Rocklin, CA: Prima Publishing, 1995.

16Muzes, G., et. al. “Effect of silimarin (Legalon) therapy on the antioxidant defense mechanism and lipid peroxidation in alcoholic liver disease (double blind protocol).” Orvosi Hetilap; 1990, 131(16): 863-866.

17Valenzuela, A. “Selectivity of silymarin on the increase of the glutathione content in different tissues of the rat.”Planta Medica; 1989, 55(5): 420-422.

18A-Z guide to drug-herb-vitamin interactions. Rocklin, CA: Healthnotes, Inc., 1999.

19Salmi, H.A. and Sarna, S. “Effect of silymarin on chemical, functional, and morphological alterations of the liver. A double-blind controlled study.” Scandinavian Journal of Gastroenterology; 1982, 17(4): 517-521.

20Jones, K. “Milk thistle may reduce some diabetics’ liver problems.” Herbs For Health; 1998, 3(2): 80.

21Velussi, M., et. al. “Long-term (12 months) treatment with an anti-oxidant drug (silymarin) is effective on hyperinsulinemia, exogenous insulin need and malondialdehyde levels in cirrhotic diabetic patients.” Journal of Hepatology; 1997, 26(4): 871-879.

22Ferenci, P., et. al. “Randomized controlled trial of silymarin treatment in patients with cirrhosis of the liver.” Journal of Hepatology; 1989, 9(1): 105-113.

23Allain, H. “Aminotransferase levels and silymarin in de novo tacrine-treated patients with Alzheimer’s disease.” Dementia and Geriatric Cognitive Disorders; 1999, 10(3): 181-185.

24Martines, G., et. al. [Silymarin in pregnancy and during hormonal contraceptive treatment. Blood chemistry and ultrastructural findings in the experimental model]. Arch Sci Med; 1979, 136(3): 443-454.

*Reprint from Hart NSP Master Reference
 Herb Allure, Inc


Milk Thistle Time- Release by Nature’s Sunshine Products

Sustained Support for Optimal Liver Health

Among the 500-plus functions the liver performs are filtering and destroying toxins in the body. To help protect the health of the liver, we need to provide it with the nutrients it requires.

Milk thistle contains the constituent silymarin, which has shown remarkable ability to help liver cells regenerate and stabilize liver cell membranes.


  • Increases the liver’s ability to filter blood.
  • Helps prevent damage from environmental toxins, alcohol and bacterial compounds.


New time-release Milk Thistle provides a consistent supply of silymarin to the liver, giving it nourishment and protection against ingested toxins.


Research shows that silymarin actually changes the structure of the outer liver cell membrane, preventing liver toxins and poisons from entering the interior of the cell. It also stimulates protein synthesis in liver cells, which generates DNA and RNA. This helps regenerate damaged liver cells. Silymarin does not stimulate growth of malignant liver tissue.

As an antioxidant, silymarin is at least 10 times more powerful than vitamin E. The level of the antioxidant enzyme, superoxide dismutase, is increased in cells with silymarin.

The German Commission E recommends milk thistle for toxic liver damage, supportive treatment in chronic inflammatory liver conditions and liver cirrhosis. Many clinical studies have examined the effects of this herb and find milk thistle highly effective with no side effects.


One tablet of NSP Time-Release Milk Thistle contains 350 mg milk thistle herb extract, providing 80 percent (280 mg) silymarin.


Take 1 tablet with a meal twice daily for all day protection.


Why you should detox, and natural ways to make it a habit

“Death begins in the colon.” Different sources attribute this quote to The Royal Society of Medicine of Great Britain, Dr. Bernard Jensen and to Elie Metchnikoff, a Russian Nobel Prize-winning biologist.

No matter who said or penned it first, it seems sensible that the organ that houses the body’s waste and toxins for elimination is the same place where adverse health conditions would originate, and, consequently, health should flourish when proper care of this organ is given.


Most of us wash our hands many times a day to remove dirt, food residue and potential bacteria. We soap and scrub our bodies regularly to remove dead skin cells, sweat, bacteria and dirt. It only makes sense to clean the inside of our bodies periodically to help remove unprocessed food material, fiber, environmental pollutants and other debris, and to expel these same items that may be stashed in cells throughout the body. This can be achieved using herbs and nutrients that strengthen the body’s regular cleansing processes and help remove or neutralize toxins. Go here to see a list of Nature’s Sunshine cleanses and detox products. 

Why Cleanse?

Just as cleaning your body’s exterior has many benefits (fresherlooking skin, removing bacteria, improving your scent), cleansing and detoxifying the body on a consistent basis offers many health benefits. These include:

  • Helps remove waste/debris and toxic chemicals (preservatives, artificial colors, pesticides, herbicides, etc.) from the colon and other body cells:
  • Improves digestion (removal of putrid material associated with gas, body odor, etc.)
  • Helps tune up the intestinal system, which has a tendency to get sluggish without proper fiber and water
  • Typically increases energy levels and promotes a feeling of well-being
  • May increase nutrient absorption
  • May increase efficiency of body systems and functions

How Toxic is Your Little Corner of the World?

You don’t have to live in a concrete metropolis to be surrounded by pollutants. Consider these sources of toxic chemicals and fumes, and steer clear where you can.

  •  Household products, including cleansers, bleaches, ammonia cleaners, and “air fresheners” that contain powerful chemicals.
  • Personal care products often contain unsafe compounds. These include some lotions, make-up, nail polish, shampoo, deodorant, mouthwash, hair spray, etc.
  • Your lawn. Putting pesticides or herbicides on your lawn can make it healthier and nicer-looking. But the treatments that kill bugs and weeds can harm your cells too.
  • Your work environment. This can include fumes from gasoline, diesel fuel, paint, thinners, industrial cleaners and such, along with seemingly harmless gases from new carpeting, furniture, etc.
  • Xenoestrogens. These chemicals mimic estrogen in the body and can throw hormone levels out of balance. They are found in plastic packaging, Styrofoam, some cleaning products and lawn treatments, cosmetics, meat, etc.

To Maintain a Toxin-Free Body, Try to Avoid These:

  • Artificial chemicals, including colorings, flavorings and preservatives
  • Medicines such as hormones, acid blockers and antibiotics
  • Polluted environments (air, water, strong chemicals and fumes, etc.)

Did You Know? During active weight loss is an ideal time to cleanse and detoxify. As your body burns up fat, it releases more stored toxins, which need to be removed before they get back into your bloodstream.

The Do’s and Don’ts of “Detox” Eating

Research shows that certain foods, nutrients and habits can greatly aid the body’s normal detoxification processes. Consider adding the following to your grocery/supplement list and start some new, clean-eating habits.

  • Fresh vegetables, including broccoli, cabbage, kale, onions, garlic, tomatoes, celery, cilantro, artichokes and olives
  • Fresh fruits, including pomegranate, papaya, berries, grapes, watermelon and cherries
  • Nuts and legumes
  • Whole grains (plenty of fiber in general)
  • Lean meats and eggs
  • Herbs and spices, including dandelion, burdock, green tea, rosemary, grape seeds, turmeric, licorice root, fenugreek, slippery elm and kelp. Note: mucilaginous herbs may help carry toxins out of the body.
  • Probiotics. When waste sits in the colon too long, levels of bad bacteria can escalate. Bacteria continue to work on waste matter, creating toxins that may leak through the colon wall and into the bloodstream. Probiotics (friendly bacteria) help rebalance ratios of good bacteria to bad in the colon. They also support digestion and the assimilation of some minerals.
  • Other nutrients: aloe, antioxidants and B vitamins
  • Plenty of water

Cleansing/detoxifying your body two to four times a year should spell excellent news for your intestinal system and promote good health, energy and focus. Show your colon some love, and it will love you right back.

How Toxic is Your Little Corner of the World?

You don’t have to live in a concrete metropolis to be surrounded by pollutants. Consider these sources of toxic chemicals and fumes, and steer clear where you can.

  •  Household products, including cleansers, bleaches, ammonia cleaners, and “air fresheners” that contain powerful chemicals.
  • Personal care products often contain unsafe compounds. These include some lotions, make-up, nail polish, shampoo, deodorant, mouthwash, hair spray, etc.
  • Your lawn. Putting pesticides or herbicides on your lawn can make it healthier and nicer-looking. But the treatments that kill bugs and weeds can harm your cells too.
  • Your work environment. This can include fumes from gasoline, diesel fuel, paint, thinners, industrial cleaners and such, along with seemingly harmless gases from new carpeting, furniture, etc.
  • Xenoestrogens. These chemicals mimic estrogen in the body and can throw hormone levels out of balance. They are found in plastic packaging, Styrofoam, some cleaning products and lawn treatments, cosmetics, meat, etc.

Sources: Detoxification 101 by Dr. Hugo Rodier, Sunshine Horizons, Jan. 2008. The Textbook of Functional Medicine 2005, 2006, The Institute for Functional Medicine





  • Blood Purifiers
  • Colon Health
  • Constipation – Sluggish Bowels
  • Detoxification
  • Digestion (improve)
  • Energy & Vitality
  • Fiber Sources
  • Halitosis (bad Breath)
  • Health & Wellness
  • Heavy Metal Poisoning
  • Immunity (increase)
  • Liver Health
  • Toxicity – Environmental Pollution
  • Weight Loss

Many experts agree that today’s society has been over-exposed to toxins such as industrial chemicals, insecticides, heavy metals, food additives and preservatives, and the residues of prescription and over-the-counter drugs. Continuous exposure to such toxins can alter metabolism, causing enzyme dysfunction, hormonal imbalance, nutritional deficiency, various psychological and neurological symptoms, and nonspecific problems such as fatigue, headache, irritability, muscle and joint pain and nausea. Fortunately, internal detoxification is one of the quickest and best ways to reverse damage from chronic exposure to toxins. Internal cleansing not only promotes general health and mental clarity, but also serves as a key factor in preventing chronic diseases associated with toxic build-up, including Alzheimer’s, arthritis, cancer, cardiovascular disease, chronic fatigue syndrome, diabetes, fibromyalgia, multiple chemical sensitivity, obesity, Parkinson’s and yeast infections. In addition, research has firmly established that dietary practices can either cause or prevent a wide range of health problems. A diet that includes fiber-rich foods (fresh fruits and vegetables, whole grains, etc.) not only offers protection against the development of chronic degenerative disease, but also provides health-promoting, therapeutic benefits. Along with antioxidants and phytochemicals, fiber-rich foods provide the body with indigestible roughage that creates bulky stools, which pass more easily and quickly through the colon. Fiber also binds to intestinal toxins, carrying them out of the body. Plus, healthy intestinal bacteria use fiber to synthesize butyric acid, which heals the mucosal cells lining the colon and also functions as an anticancer agent to prevent colorectal and other cancers.1-6

CleanStart is designed to help cleanse and detoxify the body safely and easily, while also providing 5 grams of dietary fiber in each serving. CleanStart contains herbs and nutrients to cleanse the colon of potentially harmful substances and promote healthy digestion and elimination. CleanStart’s inner-cleansing action also helps to improve nutrient absorption, enhance metabolic function, increase resistance to disease, facilitate cellular repair, and promote a greater sense of wellness and vitality. CleanStart contains 28 capsule packets and 28 fiber-rich drink packets (in either natural apple-cinnamon flavor or natural wild berry flavor), providing a 14-day supply.

Each CleanStart capsule packet contains:

Enviro-Detox is a unique formula designed to cleanse and strengthen the organs of detoxification: the intestines, kidneys, liver, lungs and skin. These organs bear the burden of filtering and eliminating toxins ingested through air-borne pollutants and exposure to chemicals, food additives, impure water, drugs and alcohol. Enviro-Detox contains burdock root, dandelion root, fenugreek seed, ginger root, pepsin, red clover flowers, yellow dock root, marshmallow root, sarsaparilla root, Lactobacillus sporogenes (a healthy colonic bacteria), cascara sagrada bark, echinacea root extract, and milk thistle seed concentrate.

LBS II is an intestinal stimulant, also referred to as a lower bowel-cleansing formula. LBS II contains a blend of herbs that activate the production of digestive fluids and bile to promote peristaltic action in the colon. LBS II relaxes abdominal cramping and spasms, enhances liver function, purifies the blood, neutralizes acidic conditions, destroys intestinal parasites and worms, and fights infection. LBS II also contains herbs that improve digestion, relieve pain and inflammation in the body, and stimulate urine flow to expel toxins and cleanse the urinary tract. LBS II contains cascara sagrada bark, buckthorn bark, licorice root, capsicum fruit, ginger root, oregon grape root, turkey rhubarb root, couch grass rhizome, and red clover flowers.

Each CleanStart drink packet contains:

Aloe vera has been approved for use by the German Commission E for occasional constipation and for conditions that require a soft stool, such as anal fissures or hemorrhoids and after rectal surgery. Taken internally, aloe vera increases peristalsis and colonic motility to improve bowel detoxification. Aloe has also been widely used by patients with inflammatory bowel disease. Internal use of aloe vera in not recommended for children, pregnant or nursing women, and patients with kidney or cardiac disease due to the potential for hypokalemia (abnormally low serum potassium levels) and potential disturbance of cardiac rhythm. Internal use of aloe vera may increase the effects of anti-arrhythmics, cardiac glycosides, loop diuretics, other potassium-wasting drugs, steroids and thiazides; thus, concurrent use should be avoided.2,7-9

Bentonite is a mineral-rich smectite clay commonly used to detoxify the intestinal tract. Bentonite both absorbs toxins into its internal structure, as well as adsorbs substances to its surface—positively charged toxins are attracted to the negatively charged edges of the clay material. Bentonite also readily absorbs water, yet remains virtually insoluble, which makes it useful as a bulk laxative.10-12

Sodium copper chlorophyllin is a mixture of water-soluble derivatives of chlorophyll, the substance that gives plants their green color. Chlorophyll has been used to eliminate bad breath and reduce the odors of infected wounds, feces and urine. Research also indicates that chlorophyll provides anti-inflammatory, antioxidant and wound-healing properties. In addition, preliminary evidence shows that chlorophyllin hinders the bioavailability of carcinogens (cancer-causing substances) and promotes their elimination in the feces.4,13-17

Malic acid is a naturally-occurring antioxidant and essential component for the production of ATP (adenosine triphosphate). ATP is the basic fuel of life, providing virtually all of the energy needed by the body. ATP is the only source of energy for cells, thus adequate levels of ATP must be maintained for normal cellular function. Malic acid also appears to be instrumental in preventing hypoxia (decreased oxygen delivery to the cells), which inhibits ATP production and causes fatigue. In addition, malic acid, which is found in apples, grapes, cranberries, and other fruits and vegetables, acts as a natural flavor enhancer.18-22

Potassium citrate, a supplemental form of potassium, is an essential mineral required for proper energy metabolism and cell function. Potassium also assists with muscle contraction and transmission of nerve signals, regulates fluid and mineral balance, and facilitates kidney function. In addition, potassium is vital for normal blood pressure—potassium deficiency may result in hypertension and heart disease. Potassium supplementation is important when cleansing or when using “potassium depleting“ diuretic drugs or laxatives.5,23,24

Psyllium hulls are a dietary fiber that have been shown to shorten gastrointestinal transit time and increase stool weight and moisture content. Thus, psyllium hulls are effective for restoring and maintaining regular and easy bowel movements. Psyllium hulls also bind with carcinogens and other potential toxins and have been shown to inhibit the growth of parasitic amoebas of the genusEntamoeba, which cause dysentery and ulceration of the colon and liver. In addition, psyllium hulls have been shown to lower serum total cholesterol and LDL cholesterol levels.25-30

Stevia, in its natural form, is considered 10-15 times sweeter than common table sugar, while extracts of stevia (in the form of steviosides) can be 100-300 times sweeter. Fortunately, most experts conclude that stevia does not affect blood sugar metabolism and may be safely used by both diabetics and hypoglycemics. And although stevia is much sweeter than sugar, it contains virtually no calories.4,31-33

Other ingredients in Apple-Cinnamon flavor: natural apple-cinnamon flavor. Other ingredients in Wild Berry flavor: natural wild berry flavor, vegetable fiber and natural carrot extract.


1Finnegan, M. “Energize Your Body!“ Energy Times; 1997, 7(3):37-40.

2Fitzgerald, F. “Detoxify for better health.“ Nature’s Impact; April/May 1998:36-41.

3Golan MD, R. Optimal Wellness. NY, NY: Ballantine Books, 1995.

4Lininger DC, S., et al. The Natural Pharmacy, 2nd ed. Rocklin, CA: Prima Health, 1999.

5Pizzorno, J & Murray, M (eds). A Textbook of Natural Medicine, 2nd ed. London: Churchill Livingstone, 1999.

6Pitchford, P. Healing With Whole Foods. Berkeley, CA: North Atlantic Books, 1993.

7″Aloe.“ American Botanical Council; 2005. <http://www.herbalgram.org/default.asp?c=aloe&gt;. Accessed January 2006.

8Langmead, L., et. al. “Randomized, double-blind, placebo-controlled trial of oral aloe vera gel for active ulcerative colitis.“ Alimentary Pharmacology & Therapeutics; 2004, 19(7):739-747.

9Fetrow, C. & Avila, J. Professional’s Handbook of Complementary & Alternative Medicines. Springhouse Corp., 1999.

10Knishinsky, R. The Clay Cure. Rocherster, VT: Healing Arts Press, 1998.

11″Bentonite.“ PDRhealth, 2003. <http://www.pdrhealth.com/drug_info&gt;. Accessed November 2003.

12Madkour, A.A., et. al. “Smectite in acute diarrhea in children: a double-blind placebo-controlled clinical trial.“ Journal of Pediatric Gastroenterology and Nutrition; 1993, 17(2):176-181.

13Ferruzzi, M.G., et. al. “Sodium copper chlorophyllin: in vitro digestive stability and accumulation by Caco-2 human intestinal cells.” Journal of Agricultural and Food Chemistry; 2002, 50(7):2173-2179.

14Nahata, M.C., et. al. “Effect of chlorophyllin on urinary odor in incontinent geriatric patients.” Drug Intelligence & Clinical Pharmacy; 1983, 17(10):732-734.

15Kumar, S.S., et. al. “Scavenging of reactive oxygen species by chlorophyllin: an ESR study.” Free Radical Research; 2001, 35(5):563-574

16Kensler, T.W., et. al. “Strategies for chemoprevention of liver cancer.” European Journal of Cancer Prevention; 2002, 11(Suppl 2):S58-64.

17Ardelt, B., et. al. “Chlorophyllin protects cells from the cytostatic and cytotoxic effects of quinacrine mustard but not of nitrogen mustard.” International Journal of Oncology; 2001, 18(4):849-853.

18Potter, B. & Orfali, S. Brain Boosters. Berkeley, CA: Ronin Publishing, 1993.

19Mindell PhD, E. Earl Mindell’s Supplement Bible. NY, NY: Fireside Books, 1998.

20″Malic Acid, Energy, & Fibromyalgia.” VRP Nutritional News; December 1995.

21Burke PhD, E. “ATP: The Energy Currency.” Nutrition Science News; March 2001.

22Paeschke PhD, T. “Dropping Calories, Maintaining Taste and Functionality.“ Food Product Design; March 2003. <http://www.foodproductdesign.com/archive/2003/0303CS.html&gt;. Accessed January 2006.

23″FDA Grants Health Claim to Potassium.” Nutrition Science News; January 2001.

24Treasure, J. & Ploth, D. “Role of dietary potassium in the treatment of hypertension.” Hypertension; 1983, 5(6):864-872.

25Herbal Medicine: Expanded Commission E Monographs. Newton, MA: Integrative Medicine, 2000.

26″Plantago ovata. (Psyllium).“ Alternative Medicine Review; 2002, 7(2):155-159.

27Moreno, L.A., et. al. “Psyllium fibre and the metabolic control of obese children and adolescents.“ Journal of Physiology and Biochemistry; 2003, 59(3):235-242.

28Jacobs, L.R. “Relationship between dietary fiber and cancer: metabolic, physiologic, and cellular mechanisms.“ Proceedings of the Society for Experimental Biology and Medicine; 1986, 183(3):299-310.

29Zaman, V., et. al. “The presence of antiamoebic constituents in psyllium husk.“ Phytotherapy Research; 2002, 16(1):78-79.

30Anderson, J.W., et. al. “Long-term cholesterol-lowering effects of psyllium as an adjunct to diet therapy in the treatment of hypercholesterolemia.“ American Journal of Clinical Nutrition; 2000, 71(6):1433-1438.

31Richard, D. Stevia Rebaudiana. Bloomingdale, IL: Vital Health Publishing, 1996.

32Gustafson, K. “Stevia.“ Natural Health; 1999, Vol. 29(6): 38.

33Curi, R., et. al. “Effect of Stevia rebaudianaon glucose tolerance in normal adult humans.“ Brazilian Journal of Medical and Biological Research; 1986, 19(6): 771-774.

Copyright 1997-2012   Herb Allure, Inc.

Herbology 101: Capsicum



Capsicum has long been used as a stimulant to increase circulation and cardiovascular activity, while at the same time lowering blood pressure and aiding in the prevention of heart attack and stroke. Capsaicin, a bitter alkaloid, is the active constituent responsible for capsicum’s ability to stimulate circulation and alter the body’s regulation of temperature, thus increasing perspiration. Capsicum’s warming properties are useful for poor circulation and related conditions, including cold hands and feet, cold stages of fevers and varicose veins.


Capsicum has been found to effectively stimulate production of digestive and mucosal fluids which helps improve digestion, soothes inflammation, enhances the removal of toxins from the body, and relieves gastrointestinal problems, including bleeding ulcers, colic, dyspepsia, flatulence and even diarrhea. Capsicum’s antiseptic properties have been proven active against various gastrointestinal pathogens (disease-causing agents).

Recent studies have found capsicum protects lung tissues from free radical damage by increasing the production of antioxidant enzymes such as SOD (superoxide dismutase).

Capsicum’s most common use is as a catalyst in herbal combinations, promoting the absorption of other nutrients. In fact, capsicum is commonly added to most any herbal formula, with the exception of some nervine or relaxing formulas. Capsicum has been proven beneficial for numerous ailments, including arthritis, cardiovascular disease, gastric ulcers, vascular headaches, impotence (resulting from venous insufficiency), infections, kidney problems, menstrual complaints, respiratory conditions such as asthma and pleurisy and thyroid dysfunction.

Capsicum is believed to help cardiovascular disease because of its stimulating action and ability to breakdown cholesterol buildup. Capsaicin, the active constituent which makes capsicum hot, promotes the conversion of cholesterol into bile acids which can then be excreted by the body—an action which may help prevent atherosclerosis.

Capsicum contains a rich supply of vitamin C and alpha-tocopherol (vitamin E), the most potent form of vitamin E with the highest nutritional and biological value. Capsicum also contains carotenes—antioxidants known for their effect in helping to prevent cancer and cardiovascular disorders and for helping to protect the body from carcinogenic and toxic chemicals. The high amount of beta-carotene in capsicum, which gives the herb its color, has also been shown to provide a healing effect on ulcers.

A French study found capsicum beneficial for chronic fatigue. Other studies indicate capsicum may be useful for mild depression and for improving athletic performance.

A small pinch of capsicum added to warm water makes an excellent gargle for hoarseness and sore throats.

Topically, capsicum was used in poultices as an antibacterial agent and as a local analgesic (pain-reliever). When capsicum is first applied it can cause a burning sensation and a degree of discomfort; however, continued exposure actually desensitizes nerve endings to pain. The alkaloid capsaicin, is responsible for this “numbing” action. Capsaicin actually blocks the neuropeptide known as Substance P, from carrying pain signals to the brain. Recent studies have shown remarkable results in the treatment of diabetic neuropathy.

Capsicum is a rich source of beta-carotene and vitamin C, as well as vitamins B1 , B2 , B3 (niacin), B5 (pantothenic acid), B6 and B9(folic acid). Capsicum also contains high amounts of cobalt and zinc.

Although large quantities of hot peppers or high doses of capsicum powder can severely irritate the esophagus, stomach, and possibly the kidneys, reports a few years ago implicating capsicum in stomach cancer or stomach ulcers have since been discounted. As a measure of safety, however, those with gastrointestinal or kidney disorders should avoid over-consumption of peppers or capsicum supplements. Also, pregnant women should avoid powdered capsicum leaves and stems, as they have been known to promote uterine contractions.

Capsicum Extract is formulated in a distilled water and grain alcohol base.



  • Circulation (improve)
  • Headache
  • Hoarseness
  • Impotence
  • Laryngitis
  • Pleurisy
  • Pregnancy – Avoid During
  • Respiratory Congestion
  • Throat – Soreness, Infection
  • Thyroid
  • Varicose Veins


 Herb Allure, Inc.

Vitamin D3



Once thought to have been conquered, vitamin D deficiency is now recognized as an epidemic in the United States. Meanwhile, epidemiologic (population) studies suggest that vitamin D deficiency is also a problem world-wide, with greater than 50% of the global population at risk for vitamin D deficiency. In fact, it is estimated that 50,000-70,000 people in the U.S. and 30,000-35,000 people in the U.K. die prematurely from cancer each year due to insufficient vitamin D.1-6


The primary source of vitamin D for both children and adults is from sun exposure, not from diet. Vitamin D is naturally produced in the skin following exposure to sunlight. In contrast, very few foods naturally contain vitamin D (namely fatty fish, egg yolks and liver) or are fortified with vitamin D. As a result, a varying percentage of people are vitamin D deficient at any time, with this percentage being higher in the winter, among the elderly or obese, in sun-deprived individuals, in dark-skinned people (heavy melanin effectively blocks skin synthesis of vitamin D), and in populations living in more poleward regions with lower sunlight exposure. Research also indicates that sunscreen inhibits the formation of vitamin D in the skin, even with extensive sun exposure. A sunscreen with a sun protection factor (SPF) of only 8 reduces the skin’s production of vitamin D by 95%. Furthermore, certain medications such as bile acid sequestrants (i.e. cholestyramine and colestipol) and anticonvulsants (i.e. phenobarbital) can cause vitamin D deficiency.2,7-18

Over the past several decades, the physiological role and importance of vitamin D has dramatically expanded, with recent findings indicating an increasing range of health benefits and therapeutic applications. Today, vitamin D is not only recognized for its crucial role in promoting bone health, but also for its emerging significance in regulating immune system function, reducing the risk of infectious and chronic diseases such as cancer, and facilitating normal brain function, central and peripheral nervous system function, blood clotting and blood cell formation, cardiac activity, and optimal muscle strength. Vitamin D also appears to play an important role in glucose metabolism—the conversion of blood sugar into energy.1,6,8,9,19-31

Perhaps the most well-known function of vitamin D is to regulate calcium absorption and metabolism for bone health. To this end, vitamin D promotes intestinal absorption of calcium and phosphorous, facilitates calcium transport, and reduces urinary calcium loss in order to keep calcium in the body and spare calcium stores in the bones. Vitamin D also promotes calcium deposition in bones and is required for the proper utilization of magnesium.6,9,12,25-29,31

However, vitamin D also exerts profound effects on human immune function. Vitamin D acts as an immune system modulator, increasing the activity of macrophages (white blood cells that destroy bacteria, protozoa and tumor cells) and providing anti-inflammatory effects. For example, recent research on the immunomodulating potential of vitamin D has shown that greater vitamin D intake is associated with both a lower risk of rheumatoid arthritis, as well as significant clinical improvement in patients treated with vitamin D. In addition, vitamin D dramatically stimulates the production of anti-microbial compounds that play a crucial role in protecting the respiratory tract against infection. Evidence from a 3-year randomized, controlled trial found that 104 women given a low dose of vitamin D (800 IU per day) were 3 times less likely to report cold and flu symptoms than an equal number of those given placebo. During the last year of the trial, a higher dose of vitamin D (2000 IU per day) was found to virtually eliminate all reports of colds and flu. Furthermore, experimental studies suggest that vitamin D may lower the risk of cancer by regulating cellular proliferation and differentiation and inhibiting angiogenesis—the formation of new blood vessels that contribute to tumor growth. In a recent randomized, controlled trial, daily intake of 1100 IU of vitamin D3 during a 4-year period was shown to dramatically reduce the occurrence of non-skin cancers. Such results corroborate other evidence indicating that higher vitamin D levels are associated with lower incidences of cancer.9,25,32-38

Vitamin D3, also known as cholecalciferol, is one of two forms of vitamin D used for nutritional supplementation. Vitamin D3 is manufactured from lanolin derived from the wool fat of sheep. The other form, known as vitamin D2 or ergocalciferol, is a vegetarian form of vitamin D manufactured by the ultraviolet radiation of yeast. Vitamin D3 has been proven to be the more potent form of vitamin D in both animal and human studies. Vitamin D3 has been shown to be at least 3 times more effective than vitamin D2 at raising serum vitamin D levels in.8,39-42

Current consensus among experts is that intakes of vitamin D between 1000 and 4000 IU will lead to more healthy serum levels of vitamin D. In fact, the physiologic requirement for vitamin D may be as high as 5000 IU per day for many individuals. For example, 4000 IU of vitamin D3 daily has been shown to safely and effectively increase serum vitamin D levels to high-normal concentrations in healthy adults. Likewise, daily oral intake of 2000 to 4000 IU of vitamin D appears to be the most effective way to improve vitamin D status in patients with congestive heart failure. Current research has also shown that the actual dietary need for vitamin D during pregnancy and lactation may be as high as 6000 IU per day and that the present recommended dietary requirement of 200 IU per day is based on little, if any, scientific or clinical evidence. In truth, high doses of vitamin D3 (6400 IU per day) have been shown to safely increase circulating serum vitamin D levels in both mothers and nursing infants, thus confirming that vitamin D intake above the currently recognized upper limit is safe by a large margin. It is important to point out that the Institute of Medicine’s present Dietary Reference Intakes for vitamin D were not designed and are not effective for preventing or treating vitamin D deficiency. Instead, government agencies designed current vitamin D intake recommendations only as guidelines to prevent particular metabolic bone diseases. Recent human clinical trials have confirmed that even prolonged daily intake of 10,000 IU of vitamin D3 is a safe tolerable upper intake level that has shown no evidence of adverse effects and is likely to pose no risk of toxicity, except in those with specific health conditions causing known hypersensitivity.9,37,43-55

Vitamin D deficiency can have serious consequences on overall health and well-being, especially considering that vitamin D status has been linked to greater disease susceptibility and/or mortality (death). In fact, vitamin D deficiency has been implicated in a host of diseases. For example, research indicates that vitamin D deficiency predisposes children to respiratory infections and is also associated with a higher risk of active tuberculosis. Growing scientific evidence has also demonstrated an association between low levels of vitamin D and a greater risk of hypertension, cardiovascular disease, depression, obesity, Type 1 diabetes, osteoporosis, cancer, and autoimmune diseases such as multiple sclerosis, rheumatoid arthritis and systemic lupus erythematosus. In addition, vitamin D deficiency is increasingly being diagnosed and treated in patients with cystic fibrosis.2,4,9,10,19,21,32,34,37,56-64

Results of several clinical and epidemiologic studies indicate an excess risk of both hypertension and diabetes mellitus in individuals with low vitamin D intake. Available data also indicate that the majority of patients with congestive heart failure have insufficient serum levels of vitamin D. In addition, vitamin D deficiency in pregnant women may be an independent risk factor for preeclampsia.48,58,65,66

Vitamin D deficiency in adults can also result in secondary hyperparathyroidism, which causes a loss of bone matrix and minerals that, in turn, increases the risk of osteoporosis and fractures. A high prevalence of low vitamin D levels has been reported in a number of populations worldwide, including women being treated for osteoporosis and those with fragility fractures—a bone fracture that occurs from a fall from standing height or less. Plus, poor mineralization of newly-formed bone matrix in adult bones causes osteomalacia—a painful bone disease that is often misdiagnosed as fibromyalgia or chronic pain syndrome or simply dismissed as depression. Vitamin D deficiency also causes muscle weakness, which increases the risk of falling and fractures.2,12,67-69

In addition, vitamin D deficiency has been found to cause dull, achy non-specific musculoskeletal pain that often persists despite pharmaceutical interventions and manual treatments. Several clinical investigations have confirmed that vitamin D deficiency is especially common among individuals with musculoskeletal pain, with some patients exhibiting severely deficient serum levels or even serum levels below the level of detection. Pain can be widespread or isolated to a particular area and is most commonly reported in the low-back and lumbar spine. In one study, 81% of female patients with chronic low back pain longer than 3 months exhibited vitamin D deficiency and significantly lower serum vitamin D levels than controls. In another study involving 267 adults undergoing outpatient treatment for chronic pain, 1 in 4 patients lacked sufficient serum levels of vitamin D. Patients with vitamin D deficiency were found to need higher doses of morphine for a longer duration, with 26% requiring nearly twice the morphine dose of those with adequate vitamin D levels. Patients with vitamin D deficiency also demonstrated overall poorer health, lower levels of physical functioning, and reported using morphine nearly twice as long as those with adequate vitamin D levels. Such results are not surprising considering that vitamin D deficiency has long been associated with pain and muscle weakness, with prior studies suggesting that pain-related symptoms associated with vitamin D deficiency respond poorly to pain medications. It is important to note that vitamin D deficiency is often misdiagnosed as fibromyalgia or chronic pain syndrome. Fortunately, high-dose vitamin D supplementation has been shown to eliminate musculoskeletal pain in a high percentage of patients with vitamin D deficiency.9,12,21,25,70-74

Mounting scientific evidence has implicated vitamin D deficiency with an increased risk of several deadly cancers. For example, epidemiologic evidence has demonstrated an inverse relationship between prostate cancer and serum vitamin D levels, suggesting that vitamin D deficiency increases the risk of prostate cancer. Likewise, observational studies indicate that inadequate vitamin D is associated with high incidence and mortality rates of breast cancer. According to the pooled analysis of two studies involving 1760 individuals, those with sufficient serum vitamin D levels (corresponding to a daily intake of 4000 IU) demonstrated a 50% lower risk of breast cancer than those with serum vitamin D deficiency. In addition, inadequate vitamin D is also associated with high incidence rates of colorectal cancer. Individuals with sufficient serum vitamin D levels or with a daily oral intake of 1000-2000 IU vitamin D3exhibited a 50% lower incidence of colorectal cancer. Death from colorectal cancer has also been shown to be inversely related to serum vitamin D levels, with high serum levels associated with a 72% less risk of death compared with lower serum levels. Furthermore, higher vitamin D indices in men with early stage non-small cell lung cancer have been correlated with higher survival rates than those with lower vitamin D indices. Higher intakes of vitamin D have also been associated with lower risks for pancreatic cancer in both men and women.1,2,6,9,19,25,75-84

Vitamin D deficiency is also a common finding among patients with inflammatory and autoimmune diseases, such as ankylosing spondylitis, multiple sclerosis, systemic lupus erythematosus (SLE), and rheumatoid arthritis. For example, research has shown that multiple sclerosis is more prevalent where environmental supplies of vitamin D are lowest (i.e. more poleward regions). Another study confirmed that patients with multiple sclerosis had significantly lower serum vitamin D levels compared to healthy controls. Likewise, patients with SLE demonstrate multiple risk factors for vitamin D deficiency, with the severity of disease correlated with lower serum vitamin D levels.33,35,85-87

Furthermore, epidemiologic studies show that higher vitamin D intake by pregnant mothers reduces asthma risk by as much as 40% in children 3 to 5 years old. However, according to a recent study in The Journal of Nutrition, pregnant women and newborns living in the northern U.S. are at high risk of having insufficient vitamin D levels, including mothers taking prenatal vitamins. In fact, another study found that the amount of vitamin D commonly provided in prenatal vitamins failed to maintain adequate maternal serum vitamin D levels, and thus supplied only extremely limited amounts of vitamin D to nursing infants via breast milk. The results of these studies indicate that higher-dose supplementation is needed to improve maternal and newborn vitamin D nutritional status. Maintaining sufficient vitamin D levels is especially important for pregnant women, since vitamin D deficiency in utero and during the first year of life is associated with a higher incidence of Type 1 diabetes. Fortunately, a study of over 10,000 infants (less than 12 months of age) who received 2,000 IU of dietary vitamin D daily showed an 80% reduction in the incidence of Type 1 diabetes, with no evidence of adverse effects.50,62,88-90

  • Asthma
  • Backache (lumbago)
  • Blood Cell Production
  • Bones
  • Brain Function
  • Calcium Deficiency
  • Cancer – General
  • Cancer, Breast
  • Cancer, Colorectal
  • Cancer, Lung
  • Cancer, Pancreas
  • Colds (common Cold)
  • Congestive Heart Failure (CHF)
  • Diabetes
  • Flu (influenza)
  • Heart Disease
  • Immunity (increase)
  • Inflammatory Conditions
  • Lactation (breast Feeding)
  • Nervous System Function
  • Osteoporosis
  • Pain
  • Pregnancy
  • Respiratory Disease, Infection

1Holick, MF. “Sunlight, UV-radiation, vitamin D and skin cancer: how much sunlight do we need?” Advances in Experimental Medicine and Biology; 2008, 624:1-15.

2—. “The vitamin D epidemic and its health consequences.” The Journal of Nutrition; 2005, 135(11):2739S-2748S.

3Park, S., Johnson, M.A. “Living in low-latitude regions in the United States does not prevent poor vitamin D status.” Nutrition Reviews; 2005, 63(6 Pt 1):203-209.

4Adams, J.S., Hewison, M. “Unexpected actions of vitamin D: new perspectives on the regulation of innate and adaptive immunity.” Nature Clinical Practice. Endocrinology & Metabolism; 2008, 4(2):80-90.

5Binkley, N., et. al. “Low vitamin D status despite abundant sun exposure.” The Journal of Clinical Endocrinology & Metabolism; 2007, 92(6):2130-2135.

6Grant, W.B., Holick, M.F. “Benefits and requirements of vitamin D for optimal health: a review.” Alternative Medicine Review; 2005, 10(2):94-111.

7Cannell, J.J., et. al. “On the epidemiology of influenza.” Virology Journal; 2008, 25;5(1):29.

8Armas, L.A., et. al. “Vitamin D2 is much less effective than vitamin D3 in humans.” The Journal of Clinical Endocrinology and Metabolism; 2004, 89(11):5387-5391.

9Vasquez, A. “Integrative orthopedics and vitamin D: testing, administration, and new relevance in the treatment of musculoskeletal pain.” Townsend Letter for Doctors and Patients; October 2004. <http://findarticles.com/p/articles/mi_m0ISW/is_255/ai_n6211963&gt;. Accessed March 2008.

10Zittermann, A. “Vitamin D and disease prevention with special reference to cardiovascular disease.” Progress in Biophysics and Molecular Biology; 2006, 92(1):39-48.

11Lamberg-Allardt, C. “Vitamin D in foods and as supplements.” Progress in Biophysics and Molecular Biology; 2006, 92(1):33-38.

12Holick, M.F. “Optimal vitamin d status for the prevention and treatment of osteoporosis.” Drugs & Aging; 2007, 24(12):1017-1029.

13Epstein, S. “The problem of low levels of vitamin D and osteoporosis: use of combination therapy with alendronic acid and colecalciferol (vitamin D3).” Drugs & Aging; 2006, 23(8):617-625.

14Hollis, B.W. “Circulating 25-hydroxyvitamin D levels indicative of vitamin D sufficiency: implications for establishing a new effective dietary intake recommendation for vitamin D.” The Journal of Nutrition; 2005, 135(2):317-322.

15Sayre, R.M., Dowdy, J.C. “Darkness at noon: sunscreens and vitamin D3.” Photochemistry and Photobiology; 2007, 83(2):459-463.

16Reichrath, J., Nürnberg, B. “Solar UV-radiation, vitamin D and skin cancer surveillance in organ transplant recipients (OTRs).” Advances in Experimental Medicine and Biology; 2008, 624:203-214.

17Hathcock, J.N. “Metabolic mechanisms of drug-nutrient interactions.” Federation Proceedings; 1985, 44(1 Pt 1):124-129.

18Torkos Phm, S. “Drug-Nutrient Interactions: A Focus On Cholesterol-Lowering Agents.” International Journal of Integrative Medicine; 2000, 2(3):9-13.

19Holick, M.F. “Vitamin D Status: Measurement, Interpretation, and Clinical Application.” Annals of Epidemiology; 2008, March 8. [Epub ahead of print]

20Roth, D.E. “Bones and beyond: an update on the role of vitamin D in child and adolescent health in Canada.” Applied Physiology, Nutrition and Metabolism; 2007, 32(4):770-777.

21Holick, M.F. “Vitamin D: importance in the prevention of cancers, type 1 diabetes, heart disease, and osteoporosis.” American Journal of Clinical Nutrition; 2004, 79(3):362-371.

22Mullin, G.E., Dobs, A.” Vitamin d and its role in cancer and immunity: a prescription for sunlight.” Nutrition in Clinical Practice; 2007, 22(3):305-322.

23Grant, W.B. “Epidemiology of disease risks in relation to vitamin D insufficiency.” Progress in Biophysics and Molecular Biology; 2006, 92(1):65-79.

24Kalueff, A.V., Tuohimaa, P. “Neurosteroid hormone vitamin D and its utility in clinical nutrition.” Current Opinion in Clinical Nutrition and Metabolic Care; 2007, 10(1):12-19.

25Mascarenhas, R., Mobarhan, S. “Hypovitaminosis D-induced pain.” Nutrition Reviews; 2004, 62(9):354-359.

26Lininger DC, S., et al. The Natural Pharmacy, 2nd ed. Rocklin, CA: Prima Health, 1999.

27Dunne, L. Nutrition Almanac, Third Edition. McGraw-Hill Publishing, 1990.

28Bergner, P. The Healing Power of Minerals, Special Nutrients and Trace Elements. Rocklin, CA: Prima Publishing, 1997.

29Spiller PhD, G. & Bruce DPH, B. Calcium: Nature’s Versatile Mineral. NY, NY: Avery, 2000.

30Tai, K., et. al. “Vitamin D, glucose, insulin, and insulin sensitivity.” Nutrition; 2008, 24(3):279-285.

31Holick, M.F. “Vitamin D: its role in cancer prevention and treatment.” Progress in Biophysics and Molecular Biology; 2006, 92(1):49-59.

32Raloff, J. “The Antibiotic Vitamin.” Science News; 2006, 170:312-317. <http://www.sciencenews.org/articles/20061111/bob9.asp&gt;. Accessed March 2008.

33VanAmerongen, B.M., et. al. “Multiple sclerosis and vitamin D: an update.” European Journal of Clinical Nutrition; 2004, 58(8):1095-1109.

34Cannell, J.J., et. al. “Epidemic influenza and vitamin D.” Epidemiology and Infection; 2006, 134(6):1129-1140.

35Cutolo, M., Otsa, K. “Review: Vitamin D, immunity and lupus.” Lupus; 2008;17(1):6-10.

36Cutolo, M., et. al. “Vitamin D in rheumatoid arthritis.” Autoimmunity Reviews; 2007, 7(1):59-64.

37 Cannell, J.J., et. al. “Diagnosis and treatment of vitamin D deficiency.” Expert Opinion on Pharmacotherapy; 2008, 9(1):107-118.

38Ali, M.M., Vaidya, V. “Vitamin D and cancer.” Journal of Cancer Research and Therapeutics; 2007, 3(4):225-230.

39Houghton, L.A., Vieth, R. “The case against ergocalciferol (vitamin D2) as a vitamin supplement.” American Journal of Clinical Nutrition; 2006, 84(4):694-697.

40“Cholecalciferol.” Wikimedia Foundation, Inc. <http://en.wikipedia.org/wiki/Cholecalciferol&gt;. Accessed March 2008.

41Trang, H.M., et. al. “Evidence that vitamin D3 increases serum 25-hydroxyvitamin D more efficiently than does vitamin D2.” American Journal of Clinical Nutrition; 1998, 68(4):854-858.

42Hart, G.R., et. al. “Measurement of vitamin D status: background, clinical use, and methodologies.” Clinical Laboratory; 2006;52(7-8):335-343.

43Ingraham, B.A., et. al. “Molecular basis of the potential of vitamin D to prevent cancer.” Current Medical Research and Opinion; 2008, 24(1):139-149.

44Bischoff-Ferrari, H.A. “Optimal serum 25-hydroxyvitamin D levels for multiple health outcomes.” Advances in Experimental Medicine and Biology; 2008, 624:55-71.

45Heaney, R.P. “Barriers to optimizing vitamin D3 intake for the elderly.” The Journal of Nutrition; 2006, 136(4):1123-1125.

46“Vitamin D and adult bone health in Australia and New Zealand: a position statement.” The Medical Journal of Australia; 2005, 182(6):281-285.

47Vieth, R., et. al. “Efficacy and safety of vitamin D3 intake exceeding the lowest observed adverse effect level.” American Journal of Clinical Nutrition; 2001, 73(2):288-294.

48Zittermann, A., et. a. “Vitamin D insufficiency in congestive heart failure: why and what to do about it?” Heart Failure Reviews; 2006, 11(1):25-33.

49Hollis, B.W. “Vitamin D requirement during pregnancy and lactation.” Journal of Bone and Mineral Research; 2007, 22 Suppl 2:V39-44.

50Wagner, C.L., et. al. “High-dose vitamin D3 supplementation in a cohort of breastfeeding mothers and their infants: a 6-month follow-up pilot study.”Breastfeeding Medicine; 2006, 1(2):59-70.

51Kimball, S.M., et. al. “Safety of vitamin D3 in adults with multiple sclerosis.” American Journal of Clinical Nutrition; 2007, 86(3):645-651.

52“Dietary Supplement Fact Sheet: Vitamin D.” National Institutes of Health. <http://dietary-supplements.info.nih.gov/factsheets/vitamind.asp&gt;. Accessed March 2008.

53Vieth, R. “Vitamin D supplementation, 25-hydroxyvitamin D concentrations, and safety.” American Journal of Clinical Nutrition; 1999, 69(5):842-856.

54—. “Vitamin D toxicity, policy, and science.” Journal of Bone and Mineral Research; 2007, 22 Suppl 2:V64-68.

55Hathcock, J.N., et. al. “Risk assessment for vitamin D.” American Journal of Clinical Nutrition; 2007, 85(1):6-18.

56Nnoaham, K.E., Clarke, A. “Low serum vitamin D levels and tuberculosis: a systematic review and meta-analysis.” International Journal of Epidemiology; 2008, 37(1):113-119.

57Michos, E.D., Melamed, M.L. “Vitamin D and cardiovascular disease risk.” Current Opinion in Clinical Nutrition and Metabolic Care; 2008, 11(1):7-12.

58Martins, D., et. al. “Prevalence of cardiovascular risk factors and the serum levels of 25-hydroxyvitamin D in the United States: data from the Third National Health and Nutrition Examination Survey.” Archives of Internal Medicine; 2007, 167(11):1159-1165.

59Wang, T.J., et. al. “Vitamin D deficiency and risk of cardiovascular disease.” Circulation; 2008, 117(4):503-511.

60Berk, M., et. al. “Vitamin D deficiency may play a role in depression.” Medical Hypotheses; 2007, 69(6):1316-1319.

61McGill, A.T., et. al. “Relationships of low serum vitamin D3 with anthropometry and markers of the metabolic syndrome and diabetes in overweight and obesity.” Nutrition Journal; 2008, 28(7):4.

62Litonjua, A.A., Weiss, S.T. “Is vitamin D deficiency to blame for the asthma epidemic?” The Journal of Allergy and Clinical Immunology; 2007, 120(5):1031-1035.

63Lansdowne, A.T., Provost, SC. “Vitamin D3 enhances mood in healthy subjects during winter.” Psychopharmacology (Berlin); 1998, 135(4):319-323.

64Stephenson, A., et. al. “Cholecalciferol significantly increases 25-hydroxyvitamin D concentrations in adults with cystic fibrosis.” American Journal of Clinical Nutrition; 2007, 85(5):1307-1311.

65Scragg, R., et. al. “Serum 25-hydroxyvitamin D, ethnicity, and blood pressure in the Third National Health and Nutrition Examination Survey.” American Journal of Hypertension; 2007, 20(7):713-719.

66Bodnar, L.M., et. al. “Maternal vitamin D deficiency increases the risk of preeclampsia.” The Journal of Clinical Endocrinology and Metabolism; 2007, 92(9):3517-3522.

67Holick, M.F. “The role of vitamin D for bone health and fracture prevention.” Current Osteoporosis Reports; 2006, 4(3):96-102.

68Jackson, C., et. al. “The effect of cholecalciferol (vitamin D3) on the risk of fall and fracture: a meta-analysis.” QJM: Monthly Journal of the Association of Physicians; 2007, 100(4):185-192.

69Epstein S. “The problem of low levels of vitamin D and osteoporosis: use of combination therapy with alendronic acid and colecalciferol (vitamin D3).” Drugs & Aging; 2006, 23(8):617-625.

70Plotnikoff, G.A., Quigley, JM. “Prevalence of severe hypovitaminosis D in patients with persistent, nonspecific musculoskeletal pain.” Mayo Clinic Proceedings; 2003, 78(12):1463-1470.

71Gerwin, R.D. “A review of myofascial pain and fibromyalgia–factors that promote their persistence.” Acupuncture in Medicine; 2005, 23(3):121-134.

72Lotfi, A., et. al. “Hypovitaminosis D in female patients with chronic low back pain.” Clinical Rheumatology; 2007, 26(11):1895-1901.

73Douaud, C. “Vitamin D deficiency linked to greater pain.” Decision News Media SAS; 2008. <http://www.nutraingredients-usa.com/news/ng.asp?id=80574&gt;. Accessed March 2008.

74Hooten, W.M., et. al. “ Prevalence and Clinical Correlates of Vitamin D Inadequacy Among Patients with Chronic Pain” from American Society of Anesthesiologists Meeting 2007. ProHealth, Inc.; 2008. <http://www.immunesupport.com/library/showarticle.cfm?id=8540&gt;. Accessed March 2008.

75Tokar, E.J., Webber, MM. “Cholecalciferol (vitamin D3) inhibits growth and invasion by up-regulating nuclear receptors and 25-hydroxylase (CYP27A1) in human prostate cancer cells.” Clinical & Experimental Metastasis; 2005, 22(3):275-284.

76Lou, Y.R., et. al. “The role of Vitamin D3 metabolism in prostate cancer.” The Journal of Steroid Biochemistry and Molecular Biology; 2004, 92(4):317-325.

77Garland, C.F., et. al. “Vitamin D and prevention of breast cancer: pooled analysis.” The Journal of Steroid Biochemistry and Molecular Biology; 2007, 103(3-5):708-711.

78Gorham, E.D., et. al. “Vitamin D and prevention of colorectal cancer.” The Journal of Steroid Biochemistry and Molecular Biology; 2005, 97(1-2):179-194.

79—. “Optimal vitamin D status for colorectal cancer prevention: a quantitative meta analysis.” American Journal of Preventative Medicine; 2007, 32(3):210-216.

80Grant, W.B., et. al. “An estimate of cancer mortality rate reductions in Europe and the US with 1,000 IU of oral vitamin D per day.” Recent Results in Cancer Research; 2007, 174:225-234.

81Jiménez-Lara, A.M. “Colorectal cancer: potential therapeutic benefits of Vitamin D.” The International Journal of Biochemistry & Cell Biology; 2007, 39(4):672-677.

82Freedman, D.M., et. al. “Prospective study of serum vitamin D and cancer mortality in the United States.” Journal of the National Cancer Institute; 2007, 99(21):1594-1602.

83Skinner, H.G., et. al. “Vitamin D intake and the risk for pancreatic cancer in two cohort studies.” Cancer Epidemiology, Biomarkers & Prevention; 2006, 15(9):1688-1695.

84Giovannucci, E. “Vitamin D and Cancer Incidence in the Harvard Cohorts.” Annals of Epidemiology; 2008, February 19. [Epub ahead of print]

85Ozgocmen, S., et. al. “Vitamin D deficiency and reduced bone mineral density in multiple sclerosis: effect of ambulatory status and functional capacity.” Journal of Bone and Mineral Metabolism; 2005, 23(4):309-313.

86Smolders, J., et. al. “Vitamin D as an immune modulator in multiple sclerosis, a review.” Journal of Neuroimmunology; 2008, 194(1-2):7-17.

87Niino, M., et. al. “Therapeutic potential of vitamin d for multiple sclerosis.” Current Medicinal Chemistry; 2008, 15(5):499-505.

88Bodnar, L.M., et. al. “High prevalence of vitamin D insufficiency in black and white pregnant women residing in the northern United States and their neonates.”The Journal of Nutrition; 2007, 137(2):447-452.

89Zipitis, C.S., Akobeng, AK. “Vitamin D Supplementation in Early Childhood and Risk of Type 1 Diabetes: a Systematic Review and Meta-analysis.” Archives of Disease in Childhood; 2008, March 13. [Epub ahead of print]

90Hyppönen, E., et. al. “Intake of vitamin D and risk of type 1 diabetes: a birth-cohort study.” Lancet; 2001, 358(9292):1500-1503.

1997-2012   Herb Allure, Inc.